White Blood Clots: "Existential Crisis For The Species Homo Sapiens"

I was calling the science in October 2021

Richard Hirschmann, embalmer, perhaps the first to point out the mysterious. elastic, white blood clots which he was removing from the veins and arteries of the deceased prior to the embalming process, and maybe the first to suggest that they were causally connected to the mass Covid vaccination program beginning 2021:

He also pointed to a very recent study:

That study is here and I would like to highlight some of the very important findings:

Anomalous, unprecedented white rubbery clots which only appeared after the vaccine rollout are the result of a chemical process, a “cascade” involving fibrin and fibrinogen.

The SOS paper has identified these white rubbery anomalous post mortem clots as “Hirschman Clots” and it points out that the biological mechanisms for their formation were elucidated in the extant scientific/medical literature going back many years:

I found one of these papers in October 2021 and pointed it out to readers:

It points out the role that fibrin and fibrinogen play in the formation of these unique clots, seen post severe Covid infection and post Covid vaccination, even before there was much talk of the rubbery white clots found by Hirschman in the US and John O’ Looney in the UK. From my article:

So what we have is a virus which, in a relatively few serious cases, mainly in older people and those with co-morbidities, causes a unique form of pro-inflammatory blood clotting which has not been observed in other respiratory diseases and in fact appears to be a phenomenon new to medicine. It is these structurally abnormal clots which are responsible for many of the fatalities associated with SARS-CoV-2 infection.

What is causing these 'structurally abnormal' blood clots? The authors identify the SARS-CoV-2 spike protein as the culprit, the mechanism which involves the binding of the spike to one of the key ingredients of blood clots, fibrin:

The central structural component of blood clots, and a key regulator of inflammation in disease, is insoluble fibrin, which is derived from the blood coagulation factor fibrinogen and is deposited in tissues at sites of vascular damage (10, 11). Hypercoagulability in COVID-19 is associated with inflammation and the formation of fibrin clots resistant to degradation despite adequate anticoagulation (3-5).

Overall, these results reveal an unanticipated role for SARS-CoV-2 Spike as a fibrinogen binding protein that alone accelerates the formation of abnormal clots with altered structure and increased inflammatory activity.

I go on to quote further from the study:

In summary, we find that SARS-CoV-2 Spike protein enhances the formation of highly inflammatory clots that are neutralized by a fibrin-targeting monoclonal antibody. Our data shed new light on the enigmatic coagulopathy found in COVID-19 revealing a causal role for fibrinogen in thromboinflammation – even independent of active viral replication. The high incidence of clotting complications in COVID-19 has been attributed to systemic inflammation (3), vascular damage including abnormal levels of circulating coagulation proteins (1, 26), genetic susceptibility to tissue factor and complement genes (27), and prothrombotic autoantibodies (28). Our findings now show that coagulopathy is not merely a consequence of inflammation. Rather, the interaction of SARS-CoV-2 Spike with fibrinogen and fibrin results in abnormal blood clot formation that in turn drives inflammation. Identification of SARS CoV-2 Spike protein as a fibrinogen binding partner provides a mechanistic basis for the formation of abnormal clots with enhanced inflammatory properties.

A vicious circle. Coagulation is not merely a result of inflammation, it causes inflammation also, a process driven by the presence of the spike protein alone and its peculiar interaction with fibrinogen and fibrin.

In October 2021, I pointed to a study which clearly showed that the SARS-CoV-2 spike protein, present in both the virus and the vaccine, was interacting with fibrin and fibrinogen to produce the novel white blood clots being observed in cadavers. In August 2024, further research based on actual chemical analysis of the clots, confirms this:

We learn that it is a lack of thrombin - necessary to convert fibrinogen into fibrin - which characterises the unique features of these clots and distinguishes them from (rare) white thrombi which form in arteries only (Hirschman clots form in veins and arteries).

The anonymous authors of the SOS study, in conclusion, state the following:

Hard to disagree. The continued coverup of these clots and the ‘died suddenly’ deaths after vaccination is almost certainly the most massive medical scandal ever and a crime against humanity which may well have very far reaching and tragic consequences,